Rousuan is a water-soluble quaternary amine, the human self by lysine (lysine) and methionine (methionine) synthesis, liver and kidneys are the main synthesizing. Nature has four amine L (levo) and right-handed (dextro) points, Rousuan no exception, but only L Rousuan have physiological activity, D Rousuan not only physiologically active, it will antagonize the L-meat the role of acid, causing physiological toxicity. Fatty acids are many organs, such as the main source of energy in skeletal muscle and cardiac muscle, fatty acids in the mitochondria through β- oxidation to produce energy; the main physiological role of L Rousuan lies in carrying long-chain fatty acids through mitochondrial inner membrane into the mitochondrial matrix (this step is required by the carnitine palmitoyltransferase I catalysis, so L Rousuan and long-chain fatty acid binding), then go through the catalytic action of carnitine palmitoyltransferase II L Rousuan separately so long chain fatty acids and long-chain fatty acids and then entering β- oxidation step, after the generation of energy. Also in the process of metabolism of L Rousuan β- oxidation and removal of membrane phospholipids resulting incomplete acyl coenzyme A (acyl-CoA) also plays an important role Hemodialysis patients during dialysis, L Rousuan will drain from the dialysate, when coupled with dietary restrictions of patients, such as meat, too little or completely vegetarian food supplement, L-Rousuan stored muscle will gradually reduce blood free meat acid (free carnitine) would be reduced, and acyl Rousuan (acyl carnitine) will be significantly increased, resulting acyl Rousuan and free Rousuan ratio (acyl carnitine: free carnitine) over 0.4, while the total amount of blood Rousuan, depending on the acyl Rousuan may be, may be normal or increased [6]. L Rousuan deficiency symptoms include unexplained lethargy, muscle weakness, low blood pressure, heart failure, arrhythmia and repeated cramps, but these symptoms are difficult to distinguish fact uremic or dialysis-related issues, or really the lack of L-Rousuan caused. The life of red blood cells may be due to increased red blood cell ATP, membrane lipid and phospholipid composition changes or pentose-phosphate shunt effect shortened disorders, and these conditions may lack L Rousuan related, and thus aggravate